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Valvojat: Jatta1001, Borrelioosiyhdistys, Bb

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Bb
Viestit: 1816
Liittynyt: Ma Tammi 26, 2009 23:13

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Viesti Kirjoittaja Bb » Pe Helmi 13, 2009 23:06

Lähettäjä: Soijuv Lähetetty: 14.11.2005 16:07

Artikkeli Samentosta borrelioosin hoidossa:

http://www.newswithviews.com/Howenstine/james26.htm

Bb
Viestit: 1816
Liittynyt: Ma Tammi 26, 2009 23:13

Viesti Kirjoittaja Bb » Pe Helmi 13, 2009 23:07

Lähettäjä: Soijuv Lähetetty: 14.11.2005 16:12

Lisää asiasta:

http://www.fourwinds10.com/news/06-heal ... sease.html
The Influence of Toxins from Bb on the Symptoms and Course of Lyme Disease

Autopsy examinations of young persons (30s) dying from what appeared to be Parkinson?s disease (PD) have frequently failed to confirm the basal ganglion damage that would be expected in classic PD seen in the elderly. Some patients with illnesses of many years? duration misdiagnosed as Amyotrophic Lateral Sclerosis, Multiple Scleroris, and Parkinson?s Disease have made incredible recoveries within periods as short as 24 to 72 hours when placed on TOA-free Uncaria tomentosa (cat?s claw) for LD. This rapid response could not rationally be attributed to improved immune function or bacteriocidal effects on spirochetes. Bb is known to produce a group of neurotoxins. The most sensible explanation for this recovery lies in turning off or blocking the neurotoxins effects of Bb on the lipid containing structures that the Bb neurotoxins are attracted to (central nervous system, peripheral nerves, muscles, joints, etc.). This sudden improvement appears to be the result of blockage and inhibition of the neurotoxins.5 The most important example of a ?Biotoxin Illness? appears to be Lyme Disease.6 Patients with symptoms of Parkinson?s Disease at a young age caused by neurotoxins would not be expected to show permanent structural destruction in the basal ganglia. These neurotoxins probably act at specific sites such as neuro-transmitters-pre and post synaptic membranes, altering dopamine, serotonin, GABA, and acetylcholine molecules, thereby blocking surface membrane receptors of various kinds which would interfere with the proper action of enzymes, coenzymes and hormones. This is only one of the damaging mechanisms of action of the neurotoxins.

The Uncaria tomentosa may have three direct beneficial effects in humans with LD:

* Immune modulation (correcting immune dysfunction).
* Direct broad spectrum anti-microbial effect on spirochetes. Quinovic acid glycosides found in TOA-free cat?s claw are similar to the quinilones widely used as antibiotics.
* Blocking the adverse neurotoxic effects on cells, enzymes, and hormones.

Whether the serious lack of energy and fatigue seen in LD are similar to the cyanate7 induced damage to the mitochondria?s ability to produce energy in the motor neurone found in amyotrophic lateral sclerosis, or is due to failure of proper calcium channel function is not clear.

Favorable Therapeutic Results with TOA-Free Cat?s Claw in Lyme Disease

A pilot study treated 28 patients with Advanced Chronic Lyme Disease with TOA-Free Uncaria tomentosa. Conventional cat?s claw contains TOA alkaloids that interfere with the desired immune modulation. The 14 person control group was given antibiotic therapy. At the study?s termination 85% of those receiving the cat?s claw preparation no longer had positive blood tests for Bb. All 28 persons had experienced a dramatic improvement in their clinical condition. No significant changes were seen in the control group. The Prima Uña de Gato can be obtained from Allergy Research Group 800-545-9960, Nutramedix (product name Samento Plus) 561-745-2917, and from Farmacopia at 800-896-1484. Dr. Whitaker?s lab can be reached by Internet at http://www.bowen.org/ or by calling 727-937-9077 to arrange blood Bb testing. Improving nutrition, detoxifying and improving mental health all contribute to good results. Removal of mercury amalgams and treatment of heavy metals may be needed.

Much of this information about LD was obtained from ?Lyme disease: Nutraceutical Breakthrough Using TOA-Free Cat?s Claw? published in Focus by Allergy Research Group (October 2003) and from the November and December 2003 issues of Dr. Robert Rowen?s Second Opinion.

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